TG6 autoimmunity and development of neurological dysfunction

  • Research type

    Research Study

  • Full title

    TG6 autoimmunity and the development of neurological dysfunction in gluten sensitivity related disorders

  • IRAS ID

    288752

  • Contact name

    Marios Hadjivassiliou

  • Contact email

    m.hadjivassiliou@sheffield.ac.uk

  • Sponsor organisation

    Sheffield Teaching Hospitals NHS Foundation Trust

  • Clinicaltrials.gov Identifier

    STH21291 , STH number

  • Duration of Study in the UK

    2 years, 0 months, 31 days

  • Research summary

    The term Gluten Related Disorders (GRD) refers to a spectrum of clinical manifestations triggered by eating gluten in individuals with gluten sensitivities. The most widely known of such disorders is coeliac disease (CD) also known as gluten sensitive enteropathy. Classical CD presents with gastrointestinal symptoms and vitamin deficiencies. Recent research demonstrates that CD can manifest with symptoms outside those related to inflammation of the gut. The most common extraintestinal manifestations present neurologically. These manifestations involve the central (CNS) and peripheral nervous systems (PNS) and can result in the characteristics of Gluten Ataxia (GA) and Gluten Neuropathy (GN), respectively.

    The existence of TG6 in the CNS and PNS and its association with neurological manifestations provides an valuable investigational area. The exact mechanisms of how TG6 affects the systems and causes the associated symptoms remains to be determined. Gluten ataxia and gluten neuropathy are devasting conditions affecting many. A diagnostic test identifying those likely to develop the neurological manifestations would enable the implementation of preventative techniques; and the diagnosis of the group presenting with idiopathic neurological symptoms that are in fact due to GRD would allow for treatment of these symptoms through a gluten free diet. Subsequently, this research has the potential to improve the quality of lives of many.

    This project aims to investigate the pathophysiology of neurological dysfunction in GRD with an emphasis on TG6 autoimmunity.

  • REC name

    North of Scotland Research Ethics Committee 2

  • REC reference

    20/NS/0117

  • Date of REC Opinion

    12 Oct 2020

  • REC opinion

    Favourable Opinion