Plasma AG:UAG ratio as a dementia biomarker

  • Research type

    Research Study

  • Full title

    Plasma acyl-ghrelin & unacyl-ghrelin (AG:UAG) ratio as a novel biomarker for dementia – a pilot study

  • IRAS ID

    250933

  • Contact name

    Jeffrey S Davies

  • Contact email

    jeff.s.davies@swansea.ac.uk

  • Sponsor organisation

    Swansea University

  • Clinicaltrials.gov Identifier

    N/A, N/A

  • Duration of Study in the UK

    1 years, 6 months, 1 days

  • Research summary

    Dementia, which includes Alzheimer’s Disease and Parkinson’s disease dementia (PDD), is the most common age-related neurodegenerative disorder, affecting more than 26.6 million people worldwide.
    Given that the population of those aged 65 or more is set to increase substantially, with 1 billion more by 2050, the burden of AD is projected to increase dramatically. Identifying an effective therapy that can delay disease onset and progression by only 1-year would reduce the number of AD cases in 2050 by 9.2 million. The estimated costs associated with dementia in the UK is £23 billion per year.
    There is no cure for dementia and diagnosis is made by clinical assessment by a neurologist. The identification of a biomarker to diagnose dementia is needed to help develop new treatments.
    The primary objective of this study will explore whether circulating levels of two stomach-derived hormones (AG and UAG) are impaired in dementia. Our current data suggests that the AG:UAG ratio is reduced in PDD. We will extend the analysis to include further patient groups that associated with cognitive impairments, namely, Parkinson's Disease - Mild Cognitive Impairment (PD-MCI) and dementia with Lewy bodies (DLB). The study will allow us to determine whether analysis of AG:UAG in blood represents a possible biomarker for dementia.

    The secondary objective is to determine whether factors from the blood of dementia patients affects the production of energy in nerve cells. This stems from the association of PD with mitochondrial dysfunction in dopamine neurones. Notably, the protective action of the hormone AG is linked to enhanced mitochondrial function. This aspect of the study will allow us to test this theory.

  • REC name

    South Central - Berkshire B Research Ethics Committee

  • REC reference

    18/SC/0538

  • Date of REC Opinion

    5 Oct 2018

  • REC opinion

    Favourable Opinion