Outcomes of surgery for unruptured aneurysm & subarachnoid hemorrhage

  • Research type

    Research Study

  • Full title

    Motor & cognitive outcomes of surgical treatment for unruptured aneurysm and subarachnoid hemorrhage

  • IRAS ID

    145257

  • Contact name

    Mark Mon-Williams

  • Contact email

    m.mon-williams@leeds.ac.uk

  • Sponsor organisation

    Leeds Teaching Hospitals NHS Trust

  • Research summary

    Aneurysmal Subarachnoid Hemorrhage (aSAH) can cause a number of cognitive and motor difficulties, which in turn may significantly impact a patient’s quality of life (e.g. Al-Khindi, Macdonald, & Schweizer, 2012). Intracranial Aneurysm Treatment (IAT) options including coiling and clipping are available, but to date there is little high quality empirical research examining the effects of aSAH on fine motor and cognitive outcomes. This is also the case in patients who have been diagnosed with an Unruptured Aneurysm (UA) but in the absence of a hemorrhage (i.e. a problem typically identified on a scan without previous history of stroke), and subsequently undergo IAT as a preventative (non-emergency) measure.

    In cases of aSAH, anecdotal reports (from patients and clinical leads) indicate that despite patients scoring well on clinical outcome measures post-operatively (which would suggest a full recovery), many patients encounter memory problems, do not return to work, and experience significant fear and anxiety as a consequence of their experience (e.g. fear of a future stroke). To date, there is also very little research surrounding the potential for IAT to yield post-operative deficits in elective ‘asymptomatic’ patients. Given the crude quality of the standardized clinical measures currently used to determine readiness of discharge and screen for deficits after IAT, it is vital to develop more robust and highly sensitive measures of post-operative neuropsychological and motor outcomes. This will allow us to (i) measure movement and cognition in patient groups with UA and aSAH; (ii) explore differences in outcomes between treatment methods (i.e. coiling vs. clipping); (iii) identify neural underpinnings of post-operative deficits by comparing neuropsychological data with structural brain changes assessed with scans obtained as a standard process during admission and follow up; (iv) design effective screening methods to support post-operative care, and inform the design of appropriate rehabilitation programmes, in the future.

    In sum, our research team proposes a longitudinal study whereby a sensitive Kinematic Assessment Tool (KAT) will be used as an objective measure of cognitive and motor domains in UA and aSAH patients that undergo IAT. The KAT is a series of touch-screen computerized tasks that have already been evidenced as providing objective measures of cognitive and motoric performance in adults and children. We will recruit a sample of patients who are matched for surgical procedure (including elective UA coiling and clipping cases, and acute aSAH coiling and clipping patients), but are otherwise fit and healthy. These patients will be tested post-operatively prior to their discharge from the ward (i.e. between 1-30 days, but will depend on the condition and IAT method) and then again at 6 weeks, 6 months and 24 months post-IAT. Our protocol will gain a more detailed understanding of patients’ post-operative abilities above and beyond what can be measured with standardized clinical tests (a selection of which will also be included at all four testing sessions). We will distinguish between the effects of surgery and the effects of the neurological condition by comparing patients who have the same IAT procedure (i.e. coil or clip) but for a different condition (i.e. UA compared to aSAH). We will explore relative outcomes of the two IAT methods by comparing patients with the same condition (i.e. UA or aSAH) but who had a different type of IAT (i.e. coil compared to clip). To explore neurological underpinnings of deficits encountered after treatment for UA or aSAH, we compare kinematic data and standard neuropsychological test scores with structural brain changes assessed with scans (i.e. MR/CT/Catheter angiogram), which are obtained as a standard process during admission and follow-up. This will allow us to make some initial predictions about the underlying neurological causes of the clinical and psychological changes seen in this patient group. All data gathered by our selection performance measures in the patient groups will also be compared with a set of age-matched controls (i.e. no UA or aSAH), recruited as part of our future studies.

    Given the paucity of research surrounding the patients’ own perspective of post-operative recovery from UA and aSAH, an optional qualitative interview (i.e. focus group) will be included, where patients will be invited to discuss from their own personal experience, any symptoms, fears and/or concerns that they encountered as a consequence of their treatment. Including this qualitative element within our project will ensure that subtle outcomes are not missed, and inform our approach to examining the motor and cognitive outcomes of UA and aSAH in any future work.

    Lay Summary of Results:

    Results
    Standardised clinical outcome measures
    Most standard measures did not differ markedly
    between sessions. Because of the categorical/ordinal
    nature of these scores, the limited change in scores
    and the small numbers of participants, it is not useful
    to examine these data using grouped statistical methods.
    Instead, we examined measures for each individual
    and compared individual scores with ceiling performance
    on the tests. There was no change in GOS between
    sessions, as nine patients received the highest classification
    (i.e. ‘Good Recovery’) at Discharge and 6/52. One
    patient was scored as having ‘Moderate Disability’
    (score¼4) at Discharge and 6/52. The pattern was similar
    for SRBI – six patients were ‘functionally independent’
    (score¼20) at both sessions, one patient scored 19
    at both sessions, and one patient scored 18 at Discharge
    and 17 at 6/52. There were also no systematic changes
    in the HADs measure. At Discharge, four of eight
    patients had ‘borderline abnormal’ or ‘abnormal’
    Anxiety scores (scores¼8, 8, 8, 12), and at 6/52, two
    scores remained unchanged as ‘borderline abnormal’,
    with one patient going from ‘normal’ to ‘abnormal’
    (6 to 16) and another from ‘abnormal’ to ‘normal’
    (12 to 6). On the Depression scale, two scores were
    ‘abnormal’ or ‘borderline abnormal’ at Discharge and
    at 6/52. Raw data for the SRBI and the HADs subscales
    are given in Table 1.
    6 Journal of Rehabilitation and Assistive Technologies Engineering
    The only standardised measures that showed systematic
    changes were the ACE-R and DSST. The ACE-R
    scores for six of eight patients improved from
    Discharge to 6/52 (mean¼93 increasing to 98) to
    scores that are near to maximum. This increase
    seemed to be driven mainly by improved Memory
    scores on the ACE-R (measuring recall, anterograde
    memory and retrograde memory), with an average
    increase from 21 to 25 out of 26 for the memory component.
    The remaining two individuals who did not
    improve on the memory element (scores at
    Discharge¼12 and 14; 6/52¼21 and 19, respectively)
    also performed poorly (i.e. scores <88, which is the cutoff
    with 94% sensitivity and 89% specificity for dementia)
    across the whole ACE-R at both testing sessions.
    Interestingly, neither of these patients fell below the
    MMSE cut-off for cognitive impairment (both patients
    scored 27 on the MMSE, which if one point less, would
    have indicated cognitive impairment). In fact, there
    were no reliable changes in the MMSE sub-test of
    the ACE-R across the whole group, with most participants
    scoring the same at both sessions. The other
    remaining ACE-R sub-tests also failed to display any
    impairment (e.g. all except one patient scored 15–16 for
    Visuospatial Ability at both sessions). Finally, DSST
    scores improved for six of 10 patients, as scores
    improved from 45 points at Discharge to 60 points
    (out of 93) by 6/52. Scores for the other four individuals
    either remained the same at both sessions (i.e. DSST
    score¼45 at both sessions for three patients) or
    declined between sessions (DSST reduced from 35 to
    25 for one patient).
    Kinematic motor tests
    Analyses of the tracking task showed patients improved
    performance accuracy from Discharge to 6/52 (F (1,
    9)¼14.20, p<.001, 2 p¼.61; mean RMSE at
    Discharge¼17.10 mm; 6/52¼12.27 mm). Accuracy
    also increased as dot-tracking speed reduced (i.e.
    ‘Slow’ tracking; F(2, 18)¼60.23, p<.001, 2p¼.87,
    "¼.53). There were no sessionspeed interactions suggesting
    performance improved between sessions similarly
    for all speeds. A single measure across speeds was
    taken to simplify further analysis. This ‘combined’ measure
    showed seven out of 10 patients exhibited ‘abnormal’
    tracking performance at Discharge (i.e. worse than
    mean healthy controls’ performanceþ95% CI;
    t(18)¼1.93, p<.05). Furthermore, despite all patients
    showing some degree of improvement, two patients still
    exhibited ‘abnormal’ tracking at 6/52 (Figure 4).
    A similar pattern was seen in the other motor tests
    (Figure 5). The steering task detected impaired accuracy
    compared to controls at Discharge (t(18)¼2.02, p<.05)
    and accuracy improved from Discharge to 6/52 (F (1,
    9)¼9.90, p<.01, 2p¼.52) – all patients improved to
    some degree; but three of 10 were still abnormal at 6/52.
    Aiming performance was only marginally worse compared
    to controls at Discharge (t(18)¼1.58, p¼.074)
    but this group performance still improved between
    Discharge (mean MT¼1.67 s) and 6/52 (mean
    MT¼1.37 s) with shorter duration movements for
    seven of 10 patients (t(9)¼2.12, p<.05), though movement
    duration was still ‘abnormal’ for two patients at 6/
    52. These findings suggest that our KAT motor tests were
    Table 1. Individual patient scores on standardised outcome measures and kinematic tests at Discharge (D) and 6/52 weeks postoperation
    (6/52).a
    SRBI HADS (A) HADS (D) CR Motor ACE-R DSST
    P Age Discharge _6/52 Discharge _6/52 Discharge _6/52 Discharge 6/52 Discharge 6/52 Discharge 6/52 Discharge 6/52
    #1 52 20 20 6 2 2 2 7 5 1.35 0.16 84 85 51 51
    #2 74 20 20 8 8 4 2 4 6 2.49 0.91 #N/A #N/A 27 27
    #3 54 20 20 12 6 8 0 7 6 0.87 0.35 92 98 38 48
    #4 72 20 20 0 0 0 0 2 6 3.38 0.14 82 82 17 30
    #5 68 20 20 8 8 3 1 5 6 1.12 0.59 94 99
    95
    56 62
    #6 31 20 20 3 2 1 1 2 9 0.99 1.34 92 43 75
    #7 55 19 19 6 16 5 8 10 13 1 0.38 100 99 64 79
    #8 49 18 17 8 7 10 9 10 16 2.32 0:23 89 97 35 25
    #9 49 #N/A #N/A #N/A #N/A #N/A #N/A 16 16 0.79 1.12 #N/A #N/A 55 55
    #10 24 #N/A #N/A #N/A #N/A #N/A #N/A 16 16 0.75 1.44 93 99 52 65
    aThe age of each participant is given, along with the following individual patient scores on (i) SRBI (max¼20); (ii) Anxiety (A) and Depression (D)
    subscales of HADs (max¼21 per subscale); (iii) maximum number of items correctly recalled (CR) out of the full 16 movement sequence comprising
    the Sequence Learning Task; (iv) a composite measure of motor performance (z-scores) on the motor task battery, including tracking, steering and
    aiming tests (i.e. a motor measure); (v) ACE-R (max¼100) and (vi) DSST (max¼93). Impaired performance measures are highlighted in red text. Dark
    grey shaded cells highlight patients with impaired CR at 6/52, and light grey shaded cells highlight those with motor difficulties at 6/52. For clarity values
    of ACE-R and DSST that are inconsistent with CR and motor impairments have been emboldened and marked with a box.
    Raw et al. 7
    sufficiently sensitive to detect improvements in performance
    between sessions, and identify individuals that had
    not yet fully recovered (through comparing performance
    with healthy controls).
    Sequence Learning Task
    Analyses of the maximum number of moves that
    patients could recall across the Sequence Learning
    Task (i.e. CR) showed that only two patients correctly
    recalled all 16 moves in both sessions (i.e. showing no
    impairment and thus no room for improvement). Four
    patients improved by 2–6 correct responses from
    Discharge to 6/52 (some data were not recorded for
    two patients at Discharge due to disruptions on the
    ward, hence initial performance could not be assessed),
    but at 6/52 there were still five patients who could only
    recall 5–6 items correctly (out of 16). Two of these
    patients also struggled with the ACE-R Memory subtests
    (patients 1 and 4 in Table 1), but the other three
    scored normally on the ACE-R (i.e. no clear cognitive
    impairment identified by the ACE-R).

  • REC name

    Yorkshire & The Humber - Leeds West Research Ethics Committee

  • REC reference

    14/YH/0009

  • Date of REC Opinion

    20 Feb 2014

  • REC opinion

    Further Information Favourable Opinion