Muscle inhibition in knee osteoarthritis phenotypes (version 1)

  • Research type

    Research Study

  • Full title

    Characterising arthrogenic muscle inhibition in different knee osteoarthritis phenotypes

  • IRAS ID

    295565

  • Contact name

    Jakob Skarabot

  • Contact email

    J.Skarabot@lboro.ac.uk

  • Sponsor organisation

    Loughborough University

  • Duration of Study in the UK

    2 years, 9 months, 30 days

  • Research summary

    People with knee osteoarthritis typically have weaker muscles that extend the knee, which are important for mobility and ambulation and thus independence and quality of life. This weakness is because of muscle inhibition, which is the reduced ability of the brain to activate all muscle fibres within the muscle. This muscle inhibition is known to cause muscle weakness and may be preventing effective muscle strengthening and rehabilitation. It has been suggested that joint pain, damage, swelling, and inflammation reduce the signal from the brain to muscle, which leads to inhibition and muscle weakness. However, the influence of these factors in patients with knee osteoarthritis has not been explored directly. Furthermore, the level inhibition is known to be variable across knee osteoarthritis phenotypes that are known to present different levels of joint damage, pain, swelling and inflammation.

    In this study, we will investigate what causes muscle inhibition in knee osteoarthritis patients and whether patients with different levels of joint damage, pain, swelling and inhibition (phenotypes) exhibit different levels of inhibition and the activation signal from the brain to muscle fibres. The level of inhibition will be measured by stimulating the nerve that activates the knee extensors during muscle contractions. During muscle contractions, we will also measure the electrical signals that control the muscle to investigate the detailed differences in the muscle activation signal. This will be done by placing electrodes on the skin covering the muscle (surface electromyography) or inserting a very thin needle electrode (26 gauge, smaller than a blood sampling needle; needle electromyography) into the muscle. Imaging techniques (magnetic resonance imaging and ultrasound) and questionnaires will be used to determine the levels of joint damage, pain, swelling and inflammation.

  • REC name

    South West - Central Bristol Research Ethics Committee

  • REC reference

    21/SW/0131

  • Date of REC Opinion

    12 Oct 2021

  • REC opinion

    Further Information Favourable Opinion