'Degen-lock' - a novel way of protecting the nervous system in MND

  • Research type

    Research Study

  • Full title

    'Degen-lock' - a novel way of protecting the nervous system in motor neuron disease

  • IRAS ID

    284217

  • Contact name

    T Newton

  • Contact email

    tim@reflectiontherapeutics.com

  • Sponsor organisation

    TMN Therapeutics Ltd

  • Clinicaltrials.gov Identifier

    Not applicable, Not applicable

  • Duration of Study in the UK

    3 years, 8 months, 15 days

  • Research summary

    TMN Therapeutics is creating a cell therapy to treat neurodegeneration. Initially we will focus our therapy on motor neurone disease (MND). In this condition motor neurons die, causing patients to experience a progressive and irreversible paralysis. Current treatments for the disease are poor, with significant side effects and only a moderate delay in symptomatic progression.

    One of the causes of neurodegeneration is neuroinflammation -- a chronic inflammation of the brain. A number of anti-inflammatory strategies has been attempted in MND research, but they have so far all failed. Strong anti-inflammatory effects suppress beneficial immune function in both the brain and the rest of the body, however, weak anti-inflammatory effects fail to be efficacious.

    A promising approach to treat neuroinflammation is co-opting part of the immune system, regulatory T cells (Tregs). These anti-inflammatory cells are able to reduce inflammation in a local environment, without shutting down beneficial inflammation in other locations. Small scale clinical trials have been carried out with Treg cell therapy in MND. In this therapy Tregs from a patient's blood are purified, grown in a laboratory environment, and large numbers re-infused back into the patient. Results are promising, however, the amount of cells required is prohibitively costly, preventing their use as a mainstream therapy.

    Our innovation is the invention of a targeting technology, 'degen-lock', which will guide Tregs to motor neurons which are suffering neuroinflammatory stress. This concentrates Tregs at sites where they are needed, greatly increasing the efficacy of therapy and reducing the number of cells needed. This reduces costs to viable levels.

    Our key objectives for this project are to establish in vivo proof-of-concept for degen-lock allowing us to apply for IP protection. Conversations with investors has indicated this is a crucial step to receive further funding.

    The experimental and legal strategy for this project has been developed since our foundation in October 2018 after extensive discussions with the Cell and Gene Therapy Catapult, patent lawyers Cooley LLC and our CRO partner, Aquila Biomedical. Aquila are based in Edinburgh and have expertise in both Treg biology and neurodegeneration, making them an ideal partner to carry out proof-of-concept work.

    Once proof-of-concept has been established for 'degen-lock' we can start developing the technology as a therapy for MND. An exciting long term plan for degen-lock is to expand into other neurodegenerative diseases such as Alzheimer's and Parkinson's once clinical success has been established in MND.

  • REC name

    North West - Preston Research Ethics Committee

  • REC reference

    20/NW/0414

  • Date of REC Opinion

    30 Oct 2020

  • REC opinion

    Further Information Favourable Opinion