Biochemical Analysis of the TOPAZ study

  • Research type

    Research Study

  • Full title

    Biochemical Analysis of the TOPAZ study

  • IRAS ID

    360703

  • Contact name

    Stuart H Ralston

  • Contact email

    stuart.ralston@ed.ac.uk

  • Sponsor organisation

    University of Edinburgh

  • Clinicaltrials.gov Identifier

    16/ES/0110 /207747, TOPAZ REC number / IRAS; 18/ES/0086 / 245197, ZIPP-LTE REC number /IRAS; 19/ES/0141 / 259285, GAPDPD REC number / IRAS

  • Duration of Study in the UK

    0 years, 11 months, 30 days

  • Research summary

    Osteogenesis imperfecta Osteogenesis imperfecta (OI) otherwise known as brittle bone disease is a rare genetic disorder characterised by increased bone fragility and multiple low trauma fractures (broken bones). A major impact of the disease is on the skeleton, OI is a systemic disorder which involves the cardiopulmonary and gastrointestinal system, soft tissues, tendons, muscle, joints, hearing, eyesight and dental health (Hald et al Calcified Tissue International 2024). While there is evidence that bone turnover is raised in OI the mechanisms are incompletely understood. A previous study showed that circulating concentrations of the biochemical marker sclerostin (SOST) were reduced in OI compared with people without OI (controls). (Nicol et al J Bone Miner Res. Feb 2018;33:307-15) In the same study SOST increased in response to teriparatide treatment which is a drug used to prevent fractures in people with osteoporosis (thinning of the bones). Other researchers have suggested that transforming growth factor beta (TGFß) is increased in OI and it has been proposed that this factor may increase bone remodelling and contribute to bone fragility in osteogenesis imperfecta (Song et al J Clin Invest. 2022;132). In this study here we propose to perform an analysis of circulating concentrations of SOST, the related cytokine Dkk1 and TGFß in stored serum samples from participants the TOPAZ clinical trial (4). We will relate the circulating concentration of these three factors to therapeutic response to treatment administered during the trial. The proposed study will clarify the role of SOST, Dkk1 and TGFß as biomarkers of disease severity and treatment response.

  • REC name

    Yorkshire & The Humber - Leeds East Research Ethics Committee

  • REC reference

    25/YH/0179

  • Date of REC Opinion

    22 Aug 2025

  • REC opinion

    Favourable Opinion